Ph.D. in Biology/Neuroscience Professor, Univ So Florida My second career focuses on cardiovascular disease I'm skeptical of LDL cholesterol as a cause of CVD
Tampa, FL
Joined December 2017
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I'm honored that my paper on statistical deception in cholesterol research will be the topic of a Grand Rounds Med School panel discussion today.
bit.ly/3Lpo5gw
@ProfTimNoakes @DoctorTro @DominicDAgosti2 @lowcarbGP @SBakerMD @ethanjweiss @davidludwigmd @DrAseemMalhotra
David Diamond retweeted
Spent a week in Portugal breaking every rule the NHS ever gave.
I ate eggs daily. I had steak. I ate local sardines, cheese and bacon. Nothing out of a packet with 47 ingredients. And I sat in the sun with no factor 50.
I slept like a baby and my nervous system had a full factory reset. No supplements, no app, just sun and real food.
Meanwhile the locals in their 80s were marching up hills I needed a sit-down after.
People talk about the Mediterranean diet like it's all vegetables and olive oil.
But the reality is grilled fish, pork, lamb, eggs, cheese, animal fat. And red wine. People walking everywhere, sitting in the sun, eating late and slowly with family, and having a nap when they felt like it. Nobody rushing, nobody grazing on "healthy snacks", nobody living on cortisol and a meal deal.
That's why they live longer. Not because someone drizzled oil on a lettuce leaf. It's the whole way they live, and you can't sell that in a bottle at the supermarket.
David Diamond retweeted
Turns out the McRib contains no rib meat. Shocker.
It actually contains shoulder, heart, stomach and intestines.
Organs are great, and everyone could use more of these, but a sandwich shaped like a rib, with "rib" in the name should probably contain some rib meat.
Don't get me started on the sauce (HFCS, soybean oil, etc.) or the bun (mono/diglycerides, soybean oil )...
nbcnews.com/business/busines…
Readers added context they thought people might want to know
The mcrib actually contains pork, water, salt, dextrose, and rosemary extract x.com/McFranchisee/s…
David Diamond retweeted
Three meta-analyses have pooled 4-9 studies to claim that low carb diets may shorten life.
Not one study examined low carb diets. Low carb was variously defined as between c. 25% and 52%! 🤣
There is no evidence from this field that low carb diets shorten life. FGS!
David Diamond retweeted
Its time to retire the 30-year-old, outdated advice that "a calorie is just a calorie" and stop fearing total food volume over nutrient density
David Diamond retweeted
The caloric equivalence of different foods is an absurdity promoted by Big Food, and sadly, most of mainstream medicine. Ditto the “eat less, move more” advice that never produces lasting weight loss.
If you’re struggling with weight and want to restore good health, come see me at GoldCare.com/dramerling.
David Diamond retweeted
David Diamond summarized his lecture on LDL/statin myths. He has familial hyperlipidemia but skips statins. Key points: extreme high LDL (FH) does not mean early death; LDL supports immunity and higher levels link to better health in elderly; coronary calcium (tied more to blood sugar than LDL) predicts risk better. High blood sugar damages LDL into harmful small-dense particles; healthy people have protective large-buoyant LDL (good trig/HDL ratio). Low-carb diets improve this profile. Statin benefits are modest, overstated statistically, and may stem from blood-thinning rather than LDL lowering; side effects (including cognition) are underplayed. He stresses the glycocalyx’s role in vessel health.
David Diamond retweeted
@grok can you please simplify this into easily understood concepts for me?
David Diamond retweeted
High LDL is not always bad. High blood sugar damages it into small dense particles (risky). Healthy people with low trig/HDL ratios mostly have large buoyant LDL (protective), even if total LDL is high.
Artery calcium and blood sugar predict heart risk better than LDL level.
Statins give only modest benefits for most people—likely from blood-thinning, not LDL lowering—and can affect cognition. Many get no benefit.
Low-carb diets shift LDL toward the healthier form.
LDL supports immunity; older adults with higher LDL often do better.
On the issue of comparing statin effects with antiplatelet meds, what I'd rather see is to compare nattokinase treatment to a statin. My prediction is that nattokinase would produce far superior outcomes compared to a statin.
@ImJustEaton @TokuHealth
Last week I delivered a lecture on LDL and statin myths to the physicians and subscribers at Goldcare.com. It is freely available here:
rumble.com/v7hgr3q-grand-rou…
I first explained that the reason why I study CVD is because 27 years ago I was diagnosed with familial combined hyperlipidemia, a rare genetic anomaly that produces high triglycerides and cholesterol which can cause premature CVD, and why I chose not to take medication for it (statins).
Then I covered the myth that most people born with extremely high LDL (familial hypercholesterolemia; FH) die young.
I then explained the importance of LDL to immune function, and why elderly people with high LDL are healthier than those that have lower LDL.
Then I showed that coronary artery calcium is much more important than LDL in CVD risk, and increased coronary calcium relates more to high blood glucose than LDL.
Then I addressed how LDL gets glycated (damaged) by high blood sugar, which causes a deterioration of LDL. The glycated LDL, referred to as small dense LDL (sdLDL) is a part of a damaged metabolism. It is the high sdLDL that explains why there is an association between LDL-C and CVD.
I then showed that high LDL-C in a healthy person is dominated by beneficial LDL, the large buoyant LDL (lbLDL). This form of LDL is found in people with a healthy ratio of triglycerides/HDL (< 2.0). This trig/HDL ratio with a low rate of events holds even for people with very high LDL-C (>170 mg/dl).
I showed the value of #lchf (low carb diet) for reducing sdLDL, increasing lbLDL and optimizing trig/HDL, which lowers the risk of CVD.
I then explained how the appearance of statin benefits have been amplified with statistical deception. The actual statins benefits are modest and most people treated yield no benefits.
There is every reason to believe that the modest benefit from statins is solely because of their anticoagulant effects, not LDL reduction. My prediction is that a head-to-head RCT of antiplatelet/anticoagulant meds vs statins would result in equal outcomes without the adverse effects of statins.
Speaking of adverse effects, I covered some of the peer-reviewed literature which has been largely ignored. A study just out demonstrated an association of adverse effects of statins on cognition:
doi: 10.3389/fphar.2026.1915633
I then covered a largely ignored but critical component of cardiovascular health: the glycocalyx. It is crucial to understand the importance of a healthy glycocalyx to understand CVD.
This talk should provide guidance for patients and physicians who have been confused and misled by those who have demonized LDL and falsely promoted statins as wonder drugs.
@markkaplan20 @dramerling @DoctorTro @SBakerMD @KenDBerryMD @lowcarbGP @zoeharcombe @DrAseemMalhotra @Fynnderella1 @DiljanMansoor @lowcarbusa @realDaveFeldman @ApoDudz @grahamsphillips @JodiNishida @drericwestman @dr_ericberg @drsimonegold
David Diamond retweeted
This was a superb talk. I encourage all who seek individualized, science-based, ethical care to join us at GoldCare.com/dramerling
Last week I delivered a lecture on LDL and statin myths to the physicians and subscribers at Goldcare.com. It is freely available here:
rumble.com/v7hgr3q-grand-rou…
I first explained that the reason why I study CVD is because 27 years ago I was diagnosed with familial combined hyperlipidemia, a rare genetic anomaly that produces high triglycerides and cholesterol which can cause premature CVD, and why I chose not to take medication for it (statins).
Then I covered the myth that most people born with extremely high LDL (familial hypercholesterolemia; FH) die young.
I then explained the importance of LDL to immune function, and why elderly people with high LDL are healthier than those that have lower LDL.
Then I showed that coronary artery calcium is much more important than LDL in CVD risk, and increased coronary calcium relates more to high blood glucose than LDL.
Then I addressed how LDL gets glycated (damaged) by high blood sugar, which causes a deterioration of LDL. The glycated LDL, referred to as small dense LDL (sdLDL) is a part of a damaged metabolism. It is the high sdLDL that explains why there is an association between LDL-C and CVD.
I then showed that high LDL-C in a healthy person is dominated by beneficial LDL, the large buoyant LDL (lbLDL). This form of LDL is found in people with a healthy ratio of triglycerides/HDL (< 2.0). This trig/HDL ratio with a low rate of events holds even for people with very high LDL-C (>170 mg/dl).
I showed the value of #lchf (low carb diet) for reducing sdLDL, increasing lbLDL and optimizing trig/HDL, which lowers the risk of CVD.
I then explained how the appearance of statin benefits have been amplified with statistical deception. The actual statins benefits are modest and most people treated yield no benefits.
There is every reason to believe that the modest benefit from statins is solely because of their anticoagulant effects, not LDL reduction. My prediction is that a head-to-head RCT of antiplatelet/anticoagulant meds vs statins would result in equal outcomes without the adverse effects of statins.
Speaking of adverse effects, I covered some of the peer-reviewed literature which has been largely ignored. A study just out demonstrated an association of adverse effects of statins on cognition:
doi: 10.3389/fphar.2026.1915633
I then covered a largely ignored but critical component of cardiovascular health: the glycocalyx. It is crucial to understand the importance of a healthy glycocalyx to understand CVD.
This talk should provide guidance for patients and physicians who have been confused and misled by those who have demonized LDL and falsely promoted statins as wonder drugs.
@markkaplan20 @dramerling @DoctorTro @SBakerMD @KenDBerryMD @lowcarbGP @zoeharcombe @DrAseemMalhotra @Fynnderella1 @DiljanMansoor @lowcarbusa @realDaveFeldman @ApoDudz @grahamsphillips @JodiNishida @drericwestman @dr_ericberg @drsimonegold
David Diamond retweeted
In 1983, America's biggest daytime talk show did something that's never happened since.
It spent an hour on a live debate on the DPT shot and vaccine mandates. Injured kids were in the studio, and the host asked what no host would dare ask today:
"Why does it have to be a law? Should we have the freedom to decide?" 🧵
Last week I delivered a lecture on LDL and statin myths to the physicians and subscribers at Goldcare.com. It is freely available here:
rumble.com/v7hgr3q-grand-rou…
I first explained that the reason why I study CVD is because 27 years ago I was diagnosed with familial combined hyperlipidemia, a rare genetic anomaly that produces high triglycerides and cholesterol which can cause premature CVD, and why I chose not to take medication for it (statins).
Then I covered the myth that most people born with extremely high LDL (familial hypercholesterolemia; FH) die young.
I then explained the importance of LDL to immune function, and why elderly people with high LDL are healthier than those that have lower LDL.
Then I showed that coronary artery calcium is much more important than LDL in CVD risk, and increased coronary calcium relates more to high blood glucose than LDL.
Then I addressed how LDL gets glycated (damaged) by high blood sugar, which causes a deterioration of LDL. The glycated LDL, referred to as small dense LDL (sdLDL) is a part of a damaged metabolism. It is the high sdLDL that explains why there is an association between LDL-C and CVD.
I then showed that high LDL-C in a healthy person is dominated by beneficial LDL, the large buoyant LDL (lbLDL). This form of LDL is found in people with a healthy ratio of triglycerides/HDL (< 2.0). This trig/HDL ratio with a low rate of events holds even for people with very high LDL-C (>170 mg/dl).
I showed the value of #lchf (low carb diet) for reducing sdLDL, increasing lbLDL and optimizing trig/HDL, which lowers the risk of CVD.
I then explained how the appearance of statin benefits have been amplified with statistical deception. The actual statins benefits are modest and most people treated yield no benefits.
There is every reason to believe that the modest benefit from statins is solely because of their anticoagulant effects, not LDL reduction. My prediction is that a head-to-head RCT of antiplatelet/anticoagulant meds vs statins would result in equal outcomes without the adverse effects of statins.
Speaking of adverse effects, I covered some of the peer-reviewed literature which has been largely ignored. A study just out demonstrated an association of adverse effects of statins on cognition:
doi: 10.3389/fphar.2026.1915633
I then covered a largely ignored but critical component of cardiovascular health: the glycocalyx. It is crucial to understand the importance of a healthy glycocalyx to understand CVD.
This talk should provide guidance for patients and physicians who have been confused and misled by those who have demonized LDL and falsely promoted statins as wonder drugs.
@markkaplan20 @dramerling @DoctorTro @SBakerMD @KenDBerryMD @lowcarbGP @zoeharcombe @DrAseemMalhotra @Fynnderella1 @DiljanMansoor @lowcarbusa @realDaveFeldman @ApoDudz @grahamsphillips @JodiNishida @drericwestman @dr_ericberg @drsimonegold
David Diamond retweeted
Colleague: "You must be missing something. There's stuff in plants you can't get from meat."
Me: "Name one."
Colleague: "Vitamin C."
Me: "Fresh meat's got it. Raw milk's got it. Nansen sat in a hole in the Arctic for nine months on bear and never lost a tooth."
Colleague: "Fibre."
Me: "Fibre's the bit you can't digest. You're listing the wrapper."
Colleague: "Antioxidants, then."
Me: "The main one's made in your own liver, out of the amino acids in last night's mutton. Nobody's ever been admitted to hospital short of blueberries."
Colleague: "Folate. That's in greens, it's named after them."
Me: "It's in egg yolk and cheese as well, and I had four of one and a slab of the other for breakfast. Named after a leaf, found in a hen."
Colleague: "Vitamin K."
Me: "The one your bones use is K2. Goat butter, cheese, yolk. The kale version mostly stays in the kale."
Colleague: "Beta-carotene."
Me: "Which you've got to convert into vitamin A, and a lot of people barely can. The yolk's already done it."
Colleague: "You're just saying egg for everything."
Me: "You're just saying leaf for everything. Mine works."
Colleague: "There has to be something only a plant makes."
Me: "There is. A chemical to stop you eating it. Every plant on earth makes one. The cow's the only one of us built to deal with them, which is why I let her go first."
Colleague: "So what's actually in your lunch?"
Me: "Mutton mince, butter, and all of the above. What's in yours?"
Colleague: "A wrap."
Me: "Then one of us is missing something."
David Diamond retweeted
Jackson had the same spine injury that left Christopher Reeves paralyzed for life. He fully recovered, stunning the world
His mom’s secret? DMSO—a simple compound vets use for spinal injuries the FDA won’t approve. But that’s not the only “incurable” neurological disorder DMSO helps.
Approximately 2000 studies support DMSO's use for conditions ranging from Alzheimer's and Parkinson's to strokes, MS, ALS, chronic pain, neuropathy, depression, epilepsy, and Down syndrome, and a physician who began treating his patients with it estimates roughly 80% of what people see neurologists for goes away with DMSO.🧵
David Diamond retweeted
Great post, Shawn.
David Diamond @LDLSkeptic quotes a study with dementia patients on statins that shows improved cognitive function after they were stopped, and then worsened after restarting them. This is as close to “proof” as you can get in medicine, but of course the study was ignored, as was the case of former astronaut Duane Graveline, who suffered global amnesia after starting statins. This resolved after stopping, but recurred when he was pushed by his wonderful doctors into restarting them!
We do not need to “prove” causation. Cholesterol is so essential for brain function that it’s synthesized by the brain, since lipoproteins are too large to get through the blood-brain barrier. Statins, especially the lipophilic ones (Lipitor) get through and block brain synthesis of this absolutely essential substance. On this basis alone they should never have been approved or prescribed. And of course, cholesterol doesn’t cause heart disease!
We are in the midst of a dementia epidemic. How many are on statins?
David Diamond retweeted
A former prime-time Fox News anchor has now revealed what happened in 2007 if a guest mentioned vaccine injuries on air.
"They would run out to the set to tell you to shut that down."
Then she explained why the network was so terrified of the topic: "Half of Fox's advertising comes from Big Pharma."
That's why you never saw these stories. 🧵
David Diamond retweeted
This is a must-watch.
Things to look for in the grocery store…including the “organic” section.
David Diamond retweeted
BREAKING 🚨: The hospital found out Gail Seiler was unvaccinated for COVID. Then they tried to MURDER her.
They STARVED her for 12 days, denied her water for 7 days, and told her husband she was going to die.
When her husband came to take her home, the hospital made her a PRISONER.
They called the police, and a 6-hour standoff followed.
When the hospital finally let them go, it wasn’t through the main entrance. It was through the freight elevator used to carry bodies to the morgue.
On the way out, a nurse told her husband, Brad, “Mark my words, she’ll be dead tonight, or before you even get home.”
Gail survived. Not because of a miracle, but because a caring physician stepped in and guided her home treatment: nebulized budesonide and ivermectin.
BRAD SEILER: “Over the next 30 days, she was weaned completely off of oxygen. Within 60 days, she had recovered sufficiently to return to work. Today, Gail is alive, healthy, active, and
enjoying life with our grandchildren and children.”
Gail’s story is horrendous, but it’s not uncommon. Hospitals were rewarded with perverse bonuses during COVID, and the more interventions they ran, the more they made.
As Attorney @RenzTom previously testified: “We have incentivized the MURDER of patients rather than incentivize TREATMENT.”
“When you go to the hospital, you get tested. They get paid more. When you get admitted for COVID, they get paid more. When they put you on remdesivir, they get paid more. When you get ventilated, they get paid more. When you die, they get paid more. This is perverse.”
David Diamond retweeted
Parents are told, “Introduce ONE new food at a time — like eggs — and wait 4–5 days to watch for ANY ALLERGIC REACTION.”
That’s official AAP & CDC guidance. Smart, cautious, and responsible.
Then at the 6-month checkup, the same experts say:
“Here’s 9 vaccines at once — DTaP, Hib, PCV, Polio, Rotavirus, HepB, and RSV. All together. No problem.”
This makes absolutely no sense...
One new food at a time with a waiting period… but a full cocktail of vaccines injected and given orally all on the same day.
Babies are tiny. Their immune systems are still developing.
Parents aren’t crazy for noticing this double standard and asking questions. We want to protect our children with real informed consent — not blind trust in the schedule.
Protect your children... say NO to vaccines.
